Guide — Pediatrics
Pediatric Respiratory Disorders
Respiratory emergencies are the leading cause of cardiac arrest in children. Nurses must rapidly distinguish between upper and lower airway disorders, recognize impending respiratory failure, and intervene before deterioration occurs. This guide covers croup, epiglottitis, bronchiolitis, and asthma.
11 min read · Pediatrics
Educational use only. Pediatric respiratory disorders vary in severity and require individualized clinical assessment. Always follow current PALS guidelines, institutional protocols, and provider orders. This guide is for nursing education and NCLEX preparation. This material supports nursing education and exam review. It is not medical advice and is not a substitute for clinical judgment, institutional policy, or medical direction. Always follow facility protocols and current provider orders.
Overview
Pediatric airways are anatomically smaller and more compliant than adult airways. Even minor inflammation or secretions can cause significant obstruction. Upper airway disorders (croup, epiglottitis) cause inspiratory stridor and may be immediately life-threatening. Lower airway disorders (bronchiolitis, asthma) cause wheezing, air trapping, and ventilation-perfusion mismatch.
The Pediatric Assessment Triangle (PAT) — appearance, work of breathing, circulation — provides the fastest initial assessment. Any child with accessory muscle use, stridor, or decreased level of consciousness requires immediate intervention.
Croup (Laryngotracheobronchitis)
Etiology:
- Parainfluenza virus (most common)
- Age: 6 months – 3 years (peak)
- Fall and winter seasonality
Pathophysiology:
- Viral inflammation of subglottic region
- Subglottic edema narrows airway
- "Steeple sign" on neck X-ray
Hallmark Assessment Findings:
- Barky, seal-like cough — classic distinguishing feature
- Inspiratory stridor — worse with crying or agitation
- Hoarse voice from laryngeal involvement
- Low-grade fever; gradual onset over 1–2 days
- Worse at night; child may appear relatively well during daytime
Nursing Priorities:
- Keep child calm — agitation worsens stridor and increases oxygen demand
- Keep parent at bedside — separation increases anxiety and airway work
- Administer racemic epinephrine (nebulized) for moderate-severe croup per order — observe for rebound (return of symptoms 2–4 hours after treatment)
- Administer corticosteroids (dexamethasone) as ordered — reduces subglottic edema
- Cool mist or humidified air — traditional management; evidence for benefit is mixed but commonly used
- Monitor for worsening stridor at rest, cyanosis, or fatigue — escalate immediately
Epiglottitis
Etiology:
- Haemophilus influenzae type B (Hib) — now rare due to vaccination
- Age: 2–7 years most common; can occur at any age
- Bacterial — rapid onset
Pathophysiology:
- Bacterial cellulitis of epiglottis and supraglottic structures
- Massive swelling can obstruct airway completely
- "Thumbprint sign" on lateral neck X-ray
Hallmark Assessment Findings (4 Ds):
- Dysphagia — inability to swallow; drooling (refuses to swallow)
- Dysphonia — muffled "hot potato" voice; NOT hoarse (distinguishes from croup)
- Drooling — unable to manage secretions
- Distress — toxic appearance, high fever (39–40°C), anxious, sitting upright in tripod position
- Sudden onset over hours (vs. croup's gradual onset)
Nursing Priorities:
- Do NOT: examine throat with tongue depressor, agitate the child, place in supine position, attempt IV access without immediate airway support available
- Allow child to remain in position of comfort — typically sitting forward, tripod position
- Keep parent at bedside to minimize agitation
- Prepare for emergent intubation or surgical airway — notify provider, anesthesia, OR immediately
- Administer IV antibiotics as ordered once airway is secured
- Provide humidified oxygen without disturbing the child
Bronchiolitis
Etiology:
- Respiratory syncytial virus (RSV) — most common
- Age: predominantly <2 years (peak infancy)
- Fall and winter seasonality
Pathophysiology:
- Viral inflammation of bronchioles → edema, mucus, bronchospasm
- Air trapping → hyperinflation → V/Q mismatch
- Small airways particularly vulnerable in infants
Assessment Findings:
- URI symptoms precede lower airway involvement (rhinorrhea, low-grade fever)
- Expiratory wheezing — lower airway obstruction
- Tachypnea, intercostal retractions, nasal flaring
- Poor feeding — hypoxia and tachypnea impair coordinated suck-swallow
- Crackles on auscultation; prolonged expiratory phase
- SpO₂ may drop, especially during feeding
Nursing Priorities:
- Supportive care is primary — bronchiolitis is viral; antibiotics are not indicated
- Maintain airway patency — bulb suction or nasopharyngeal suction to clear secretions
- Supplemental oxygen to maintain SpO₂ ≥90–95%
- Small, frequent feedings or NG feeding if respiratory distress impairs oral feeding
- Monitor hydration status — tachypnea increases insensible losses
- High-flow nasal cannula (HFNC) or CPAP for moderate-severe cases
- Palivizumab (Synagis) prophylaxis for high-risk infants (premature, chronic lung disease, congenital heart disease)
Pediatric Asthma
Etiology / Triggers:
- Chronic inflammatory airway disease
- Triggers: allergens, URI, cold air, exercise, smoke, stress
- Most common chronic pediatric disease
Pathophysiology:
- Bronchospasm + mucosal edema + mucus hypersecretion
- Air trapping → hyperinflation → barrel chest (chronic)
- Expiratory airflow obstruction
Assessment Findings — Acute Exacerbation:
- Wheezing, dyspnea, chest tightness, cough (especially nocturnal)
- Tachypnea, tachycardia, accessory muscle use
- Silent chest = severe obstruction — no air movement; immediately life-threatening
- Peak expiratory flow rate (PEFR) <50% predicted = severe attack
- Hypoxia: SpO₂ <92% = severe; cyanosis = impending respiratory arrest
Nursing Priorities:
- Position upright (High-Fowler's) to maximize diaphragm excursion
- Supplemental oxygen for SpO₂ <94%
- Administer short-acting beta-2 agonist (albuterol) via nebulizer or MDI with spacer — first-line for acute exacerbation
- Ipratropium (anticholinergic) added for moderate-severe attacks
- Systemic corticosteroids for moderate-severe attacks — reduce inflammation
- IV magnesium sulfate for severe refractory attacks in the ED setting
- Teach proper inhaler technique with spacer for all ages; peak flow monitoring
- Identify and document triggers; develop asthma action plan
NCLEX Pearls
- Croup vs. Epiglottitis: Barky cough + hoarse = croup; Drooling + muffled voice + tripod = epiglottitis
- Never examine the throat of a child suspected to have epiglottitis — can trigger complete airway obstruction
- Bronchiolitis management is supportive only — no routine bronchodilators or corticosteroids
- Silent chest in asthma = ominous sign; the child is too obstructed to move air enough to wheeze
- Respiratory failure causes cardiac arrest in children — always address airway first
- Racemic epinephrine rebound: symptoms return 2–4 hours after treatment — observe child for at least 3–4 hours
- Albuterol (SABA) = rescue inhaler for acute symptoms; ICS = controller for daily prevention
Related Resources
Standards & sources
Fact-checked Jun 21, 2026This page is written to align with American Academy of Pediatrics (AAP) · CDC / ACIP (immunization schedule). It is an educational summary, not a citation of any single document — always verify specific doses, values, and protocols against current guidelines and your facility policy. How we source content →
