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Apex Nursing

Chart — Cardiac

EKG Rhythm Chart

Bedside recognition reference for 14 common cardiac rhythms — rate, regularity, P wave, PR interval, QRS criteria, and key identification features with clinical significance and immediate nursing priorities.

Educational use only. Always assess the patient before acting on monitor findings. Rhythm identification requires clinical correlation. Follow your institution’s protocols and scope of practice. This material supports nursing education and exam review. It is not medical advice and is not a substitute for clinical judgment, institutional policy, or medical direction. Always follow facility protocols and current provider orders.

NormalMonitorUrgentArrest— Clinical urgency tier

Rhythm Recognition Reference

RhythmRate (bpm)RegularityP WavePR IntervalQRSKey Recognition Feature
Normal Sinus Rhythm (NSR)Normal
60–100RegularUpright in lead II; 1 P per QRS0.12–0.20 s< 0.12 s (narrow)All five criteria met — the baseline normal rhythm
Sinus BradycardiaMonitor
< 60RegularUpright in lead II; 1 P per QRS0.12–0.20 sNarrowLooks like NSR — rate is the only difference
Sinus TachycardiaMonitor
100–160RegularPresent; may be buried in preceding T wave at fast ratesNormal (may shorten slightly)NarrowNSR pattern — just faster; always has an underlying cause
Atrial FibrillationUrgent
Atrial: chaotic; Ventricular: 60–160+Irregularly irregularAbsent — chaotic fibrillatory baselineAbsentNarrow (usually); wide if aberrant conductionNo two R-R intervals are equal — no pattern to the irregularity
Atrial FlutterUrgent
Atrial: 250–350; Ventricular: depends on AV ratioUsually regular; variable if AV ratio variesSawtooth flutter waves (F waves) at ~300/minVariable — determined by flutter:QRS ratioNarrowClassic sawtooth in II, III, aVF, V1; 2:1 ratio gives ventricular rate ~150
SVT (Supraventricular Tachycardia)Urgent
150–250RegularHidden in QRS or T wave; retrograde (inverted) if visibleNot measurableNarrow (unless aberrant conduction)Abrupt onset and termination; regular narrow tachycardia
First-Degree AV BlockMonitor
60–100RegularPresent; 1 P per QRS> 0.20 s (prolonged)NarrowAll P waves conduct — just delayed; PR > 1 large box (5 small squares)
Second-Degree AV Block Type I (Wenckebach)Monitor
VariableIrregular (grouped beating pattern)Present; more P waves than QRS complexesProgressively lengthens → then one P wave is blocked (no QRS)NarrowPR gets longer, longer, longer — then a P wave with no QRS (dropped beat)
Second-Degree AV Block Type IIUrgent
Variable (slower due to dropped beats)Regular until dropped beat; then irregularPresent; more P waves than QRS complexesConstant — then suddenly a P wave is not conducted without warningNarrow or wide (often bundle branch block)Fixed PR — unexpected dropped beat; no lengthening pattern
Third-Degree (Complete) AV BlockUrgent
Atrial: varies; Ventricular: 20–60Both P and QRS regular — but independent of each otherPresent; firing at own rate; NO relationship to QRSNo consistent PR — constantly changesWide (ventricular escape) or narrow (junctional escape)Complete AV dissociation — P waves and QRS complexes march through independently
Ventricular Tachycardia (VT)Arrest
100–250RegularAbsent or AV dissociationNot measurable≥ 0.12 s (wide, bizarre morphology)Wide bizarre complexes at fast rate — immediately assess for pulse
Ventricular Fibrillation (VF)Arrest
No measurable rateChaotic — no patternAbsentAbsentNo organized QRS complexesDisorganized chaotic waveform with no identifiable QRS
Pulseless Electrical Activity (PEA)Arrest
Organized electrical activity visible on monitorVaries — depends on underlying rhythmMay be presentVariesVaries — may look near-normalOrganized electrical activity on monitor — NO palpable pulse
AsystoleArrest
NoneNoneAbsentAbsentAbsentFlat line — confirm in 2 leads; do not treat fine VF as asystole

Clinical Significance & Nursing Priorities

RhythmClinical Significance & Priorities
Normal Sinus Rhythm (NSR)Normal cardiac conduction. Document as baseline. Any deviation requires identification.
Sinus BradycardiaMay be normal (athletes, sleep, vagal tone). Treat only if symptomatic: hypotension, altered LOC, syncope → atropine 1 mg IV q3–5 min (max 3 mg).
Sinus TachycardiaAlways reactive. Identify and treat the cause: pain, fever, hypovolemia, anxiety, sepsis, hypoxia, medications. Treating tachycardia without addressing cause is dangerous.
Atrial FibrillationEmbolic stroke risk — assess anticoagulation status. Determine rate vs. rhythm control strategy. New-onset AFib with hemodynamic instability → cardioversion.
Atrial FlutterSimilar stroke risk to AFib. 2:1 AV conduction (most common) produces ventricular rate ~150 bpm — easily missed as sinus tachycardia. Look for hidden F-waves.
SVT (Supraventricular Tachycardia)Usually AVNRT (most common). Vagal maneuvers first; adenosine 6 mg IV if vagal fails. Unstable SVT → synchronized cardioversion. Often self-terminating.
First-Degree AV BlockUsually benign. Every P wave still conducts. Monitor for progression to higher-degree block. May be caused by increased vagal tone, beta-blockers, digitalis, or myocarditis.
Second-Degree AV Block Type I (Wenckebach)Block occurs at AV node. Usually benign and reversible. Grouped beating pattern is characteristic. Monitor for progression to Type II or complete block.
Second-Degree AV Block Type IIBlock occurs below AV node (His-Purkinje). Higher risk than Type I — can progress suddenly to complete heart block. Pacemaker often required. Notify provider immediately.
Third-Degree (Complete) AV BlockMedical emergency. Atria and ventricles controlled by independent pacemakers. Hemodynamic compromise is common. Transcutaneous pacing preparation and immediate provider notification required.
Ventricular Tachycardia (VT)Check pulse immediately. With pulse + stable: amiodarone IV. With pulse + unstable: synchronized cardioversion. Pulseless VT: CPR + defibrillation (treat as VF).
Ventricular Fibrillation (VF)Cardiac arrest. Immediately: CPR → defibrillation → CPR → epinephrine → amiodarone. Coarse VF defibrillates more reliably than fine VF. Confirm in 2 leads before treating fine VF as asystole.
Pulseless Electrical Activity (PEA)Cardiac arrest. CPR immediately. Identify and treat reversible causes: H's (hypovolemia, hypoxia, H+, hypo/hyperkalemia, hypothermia) and T's (tension pneumo, tamponade, toxins, thrombosis).
AsystoleCardiac arrest with poorest prognosis. CPR immediately + epinephrine 1 mg IV q3–5 min. Confirm true asystole in 2 leads — fine VF can look like asystole and is defibrillatable.

AV Block Quick Comparison

1st Degree

All P waves conduct. PR only prolonged. Benign.

2nd Degree Type I (Wenckebach)

PR lengthens progressively → dropped beat. Block at AV node. Usually benign.

2nd Degree Type II

Fixed PR → sudden dropped beat (no warning). Block below AV node. Higher risk. May need pacing.

3rd Degree (Complete)

No P-to-QRS relationship. Complete AV dissociation. Medical emergency — pacing required.

Cardiac Arrest Rhythms — ACLS Overview

Shockable

VF and pulseless VT → CPR + defibrillation

Non-Shockable

PEA and Asystole → CPR + epinephrine; address reversible causes (H’s & T’s)

In all arrest rhythms: call for help, initiate CPR, apply defibrillator pads, establish IV/IO access, and follow ACLS algorithm.

Related Resources

Standards & sources

Fact-checked Jun 20, 2026

This page is written to align with AHA / ACC ECG Standards / ACLS Guidelines. It is an educational summary, not a citation of any single document — always verify specific doses, values, and protocols against current guidelines and your facility policy. How we source content →